Its action in the male body is like that of LH, stimulating the Leydig cells in the testes to produce testosterone even in the absence of endogenous LH. HCG is therefore used during longer or heavier steroid cycles to maintain testicular size and condition, or to bring atrophied (shrunken) testicles back up to their original condition in preparation for post-cycle Clomid therapy. This process is necessary because atrophied testicles produce reduced levels of natural testosterone, this situation should be rectified prior to post-cycle Clomid therapy.
The fact that Clomid is an effective HPTA and endogenous Testosterone stimulating agent is well documented and has been covered extensively thus far throughout this profile. The fact that Clomid is less effective at acting as an Estrogen antagonist in breast tissue compared to its effectiveness in restoring hormonal function is the reason as to why it is utilized primarily and exclusively for the latter reason among anabolic steroid users. However, it is still important to note that on a mg for mg basis, Nolvadex is far superior to Clomid. Normally it is because of this that if any individuals still insist on the use of Clomid at all, it should be included as a multi-compound PCT program in which it is also used with Nolvadex and perhaps HCG (many will also include the use of an aromatase inhibitor to mitigate the Estrogenic effects of HCG administration).